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When introduced into the body, these cells direct themselves toward the brain, exploiting specific chemical signals (cytokines) and chemokines released in damaged areas

Some low molecular weight compounds are able to penetrate the BBB with the help of specific transporters, for example, the large neutral amino acid transporter 1 (LAT1) for L-DOPA [128130 ] or the efflux transporter P-glycoprotein (P-gp) for lipophilic drugs [131, 132]

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Before advancing these strategies to clinical application, several key challenges must be addressed, such as the complexity and functional redundancy of the ferroptosis regulatory network, the limitations of existing animal models in simulating the pathological features of human chronic OA, and the lack of reliable biomarkers for assessing intra-articular ferroptosis activity in humans