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Notably, the unique role of PPARa as a transcriptional hub for metabolic regulation provides mechanistic justification for its ferroptosis-modulating capacity [22, 23]

Current Medicinal Chemistry, 19(23), 4000-5

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found that inhibiting Gln uptake increases the sensitivity of ovarian cancer cells to paclitaxel by blocking the activation of the mTORC1/S6K signaling pathway, downregulating Mcl-1 levels, and increasing Bcl-2 phosphorylation (96)